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M. Albert Basson

Professor M. Albert Basson

Professor of Developmental Neurobiology

Biography

Professor Basson obtained his PhD from the University of Cambridge and underwent post-doctoral training at the National Institute for Medical Research (Mill Hill), Mount Sinai School of Medicine in New York, King’s College London and UCSF.

He is Professor of Developmental Neurobiology in the Centre for Craniofacial and Regenerative Biology and the MRC Centre for Neurodevelopmental Disorders (https://devneuro.org/cndd/aboutus.php). His research has provided insights into the causes of developmental defects of the thymus, kidney, cardiovascular system and cerebellum and has been published in top international journals.

The current focus of the Basson laboratory (http://bassonlab.com/) is to uncover the function of chromatin remodelling factors in brain development and to understand how mutation of these factors cause neurodevelopment disorders and intellectual disability. They have identified key roles for the chromatin remodelling factor CHD7, mutated in CHARGE syndrome, in development of the cerebellum and gene dosage-sensitive roles for the autism-associated factor CHD8 in development of the neocortex.

Teaching: Epigenetics, Neurodevelopmental Disorders

    Research

    KCL_Dentistry2010_MG_5469
    Basson Lab

    Research that is primarily focused on chromatin remodelling factors implicated in neurodevelopmental disorders.

    News

    New recessive neurodevelopmental disorder identified

    Academics at King's have reported a rare neurodevelopmental condition characterised by intellectual disability, ataxia with cerebellar hypoplasia and delayed...

    Prdm-13-image

      Research

      KCL_Dentistry2010_MG_5469
      Basson Lab

      Research that is primarily focused on chromatin remodelling factors implicated in neurodevelopmental disorders.

      News

      New recessive neurodevelopmental disorder identified

      Academics at King's have reported a rare neurodevelopmental condition characterised by intellectual disability, ataxia with cerebellar hypoplasia and delayed...

      Prdm-13-image